Recently, Csorba et al. reported that anti-C1q antibodies specific for the major linear epitope of CLR are cross-reacting with EBNA-1 (Epstein–Barr virus nuclear antigen 1)-derived peptide, implying that anti-C1q in SLE could be induced through molecular mimicry by EBV [36]. This evidence concerns the gene DCLK3 and systemic lupus erythematosus.