Bcr-abl has been also reported to drive the abnormal proliferation of cancer cells through the constitutive activation of multiple oncogenic signaling pathways such as Mitogen-activated protein kinase kinase/ extracellular-signal-regulated kinase (MEK/ERK), PI3K, and Janus kinase/Signal transducer and activator of transcription (JAK/STAT) signaling pathways [13,14,15,16,17]. Here, SOAT1 is linked to cancer.