Since the expression of the mature miRNAs transcribed from the cluster miR-199a~214 was strongly upregulated by TGFβ1 in FAPs (Figure 2C) and also altered in DMD (Figure 1C), we tested the effect of LNA-antisense oligonucleotides (ASOs) directed against the three most-expressed mature miRNAs of the cluster (LNA-199a-3p, LNA-199a-5p or LNA-214-3p) on FAP differentiation. The gene discussed is FAP; the disease is Duchenne muscular dystrophy.