ARG1 and infection: In vivo experiments with these mice have previously shown that the deletion of ARG1 in hematopoietic and endothelial cells improved the outcome of infections with Leishmania major, Mycobacterium tuberculosis and Trypanosoma brucei [10,13,14], whereas in our model of murine salmonellosis no reduction in the pathogen load was detected in liver, spleen and blood.