Given that EPLIN participates in the regulation of ciliation progression, interacting with LUZP1 [32], and that depletion and dysfunction of cilia can result in diseases such as blindness, cystic kidneys, etc. [32,34], this demonstrates EPLIN’s implication and importance in other biological processes, not only in carcinogenesis and tumour development. This evidence concerns the gene LIMA1 and neoplasm.