We found PKA-mediated phosphorylation sites of cTnI at sites S23/S24 and S43 mouse canonical sequence, which correspond to S42 (human canonical sequence), to be significantly reduced in HF compared to the donors (Figure 2H,I), whereas PKC-mediated cTnI phosphorylation at T143 was unchanged (Figure 2J). The gene discussed is PRRT2; the disease is hydrops fetalis.