Due to the fact that AT1R antagonists and Ang 1-7 proved to be promising in preventing eosinophilic infiltrates and airway hyperactivity in animal models [58], as well as moderating allergic asthma [59], it is to be expected that ACE2 will find its place in the pathophysiology of asthma, given the fact that ACE2 levels in the sputum of asthma patients are greater than those of the healthy population [63]. The gene discussed is ACE2; the disease is asthma.