Both the aforementioned ZEB2 presence in the prospective basal ganglia of the forebrain (including the MGE, LGE and CGE), but also phenotypic analysis of the Nestin-Cre;Zeb2-cKO used by Seuntjens and co-workers (2009 [47]; see above), as well as seizures and epilepsy in MOWS patients, then prompted investigation of ZEB2 in interneuron generation in the ventral telencephalon and in migration of these cells [176]. The gene discussed is ZEB2; the disease is epilepsy.