Some of these new roles could prompt clinical geneticists to follow up MOWS patients longitudinally in new directions, for example with regard to remyelination by Schwann cells [67,68] and in epilepsy, caused by ZEB2 deficiency and its effects on guided migration in GABAergic interneurons [64] and their fate in the forebrain [63]. This evidence concerns the gene ZEB2 and epilepsy.