Because NAFLD phenotypes including fibrosis severity [25] and hepatic fat [26] have previously shown mild association with levels of the inflammatory marker C-reactive protein (CRP), we compared our results to those of a large (N = 8863) EWAS investigating the association between CRP and DNAm in blood [27] to examine whether our results could reflect an inflammatory signature of DNAm. This evidence concerns the gene CRP and metabolic dysfunction-associated steatotic liver disease.