In the cardiovascular system, iron overload has been reported to augment angiotensin II (Ang II)‐induced cardiac fibrosis,10 aggravate atherosclerosis,11, 12 induce vascular dysfunction in resistant pulmonary arteries associated with right ventricular remodeling,13 accelerate thrombus formation after arterial injury,14 increase vascular oxidative stress, and impair vasoreactivity.15 Here, AGT is linked to atherosclerosis.