LEP and bacterial urinary tract infection: We applied an approach that we named normalized endpoint work through (NEW) and used principal components analysis (PCA) and hierarchical clustering to show that the VSA phenome of 6‐ to 10‐week‐old male mice with diabetic diuresis (driving by a leptin mutation, Lepob/ob, in BTBR mice), irritative voiding from UTI (intraurethral E. coli UTI89) and bladder outlet obstruction (driving by slow‐release, subcutaneous testosterone, and estradiol implants) are fairly unique.