ATG7 and Pulmonary hemorrhage: Since memory B cells are potent antigen presenting cells and autophagy could regulate antigen presentation (16, 17, 73), it is possible that defective antigen presentation by autophagy-deficient memory B cells resulted in defective amplification of autoimmune responses, thus leading to the reduced susceptibility of B/Atg7–/– mice to pristane-induced pulmonary hemorrhage.