Dampening of lung fibrosis is also being investigated as a means of aiding COVID-19 recovery/minimising COVID-19-induced damage [97]; as AR is expressed in lung fibroblasts [56], and as we know from the prostate, AR expressed in fibroblasts modifies fibrotic activity [98], so this too may be a means by which anti-androgens could benefit COVID-19 patients. The gene discussed is AR; the disease is COVID-19.