TP53 and Leber congenital amaurosis: The results showed that exosomal miR-218-5p and miR-363-3p from endothelial progenitor cells promoted angiogenesis, proliferation, and mesenchymal-endothelial transition of CFs by targeting the p53 promoter and JMY 3′UTR, respectively, which further alleviated LCA-induced chronic myocardial infarction.