ACE2 and infection: Thus, it can be hypothesized that infection of the fetus does not occur because of the reduced coexpression and colocalization of placental angiotensin-converting enzyme 2 and transmembrane serine protease 2, and that the immunosuppression present during pregnancy could protect not only most women from the cytokine storm, massive immune activation, and hyper inflammation, but also the fetus from a possible maternal immune aggression.