Compared with Sham‐operated mice, AAA lesions in WT mice exhibited an increased mRNA expression of KLF4 (synthetic VSMC phenotype marker) and lower levels of ACTA2 and SM22α (contractile VSMC markers) (Figure 4E), alongside upregulated M1 pro‐inflammatory macrophage markers (ARG2 and iNOS) and downregulated M2 reparative genes (ARG1 and CD206) (Figure 4F). The gene discussed is KLF4; the disease is triple-A syndrome.