Autophagy: H19 overexpression suppressed autophagy of glioma cells via regulating the mammalian target of rapamycin (mTOR)/Unc-51 like autophagy activating kinase 1 (ULK1) axis by inducing increased ULK1 phosphorylation and inhibiting mTOR phosphorylation (53). Here, H19 is linked to glioma.