COVID-19 results in an intense inflammatory response that is dependent on several mediators, including TNF and IL-6.[8] Cytokine storm can result in severe pulmonary inflammation.[9] Many attempts to modulate this erratic immune response have been described, but these have shown little success.[10,11] A recent publication reported that SARS-CoV-2/M. leprae coinfection was followed by intense IL-6- and IL-12-dependent responses, but the clinical presentations of both diseases were not changed.[4]. This evidence concerns the gene IL6 and inflammatory response.