SCDAL and liver cancer: LncRNAs have been proposed to act as scaffolds that coordinate distinct chromatin‐modifying complexes to target DNA loci.[27] For example, lncBRM interacted with BRM (the ATPase subunit of SWI/SNF complex) to activate YAP1 signaling and promote self‐renewal of liver cancer stem cells, and the binding fragment was predicted to form stable stem‐loop structure.[28] The binding fragments of SCDAL and SNF5 are predicted to harbor stable stem‐loop structures which may facilitate the binding of SNF5 to the promoter of GDF6.