Although the involvement of these proteins in GBS pathogenesis was not investigated in the current study, previous studies have reported that BspA and BspC are critical adhesins in GBS, which interact with the gp340 protein on human epithelial cells and the host cytoskeleton component vimentin, thereby contributing to GBS meningitis pathogenesis (65, 66). Here, VIM is linked to Guillain-Barre syndrome.