PSP is a form of atypical parkinsonism pathologically characterised by the intracellular aggregation of 4‐repeat (4R) tau protein in the form of neurofibrillary tangles, neuropil threads, oligodendroglial coiled bodies and tufted astrocytes in the subcortical and cortical regions of the brain, resulting in neurodegeneration [92, 93]. The gene discussed is MAPT; the disease is supranuclear palsy, progressive, 1.