Taken together, these findings indicated that the prolonged unjammed phase induced by EGFR and YAP in IPF epithelia is likely not due to other downstream EGFR targets (i.e., AKT1/2/3, STAT3, JAK1/2, PCK), but instead is specific to the ERBB-YAP axis. This evidence concerns the gene EGFR and idiopathic pulmonary fibrosis.