Like lymphocytes and macrophages, they play an active, reciprocal role in the context of cancer: tumours can induce elevated production of neutrophils in the bone marrow as well as recruit them to the site,24, 25 where they will then be polarised towards pro‐tumour and metastasis‐promoting phenotypes through TGFβ‐ and G‐CSF‐dependent mechanisms.26, 27, 28, 29. This evidence concerns the gene CSF3 and neoplasm.