Potentially regulates RAD51 through HDAC2 in some cancers; maintains chromatin state; chemosensitizer; tumor suppression; increases tumor antigen presentation; cell cycle progression; suppresses cell proliferation; induces apoptosis through the upregulation of miR-31 (pro-apoptotic microRNA) (Briere et al., (2018); Q. Zhang et al., (2016b; Mondal et al., (2020); Headley et al., (2019); Shan et al., (2017); Yan and Efferth (2020). Here, HDAC2 is linked to neoplasm.