THY1 and Lewy body dementia: Another possible explanation could be the loss of inhibitory input, circuitry remodeling, and hippocampal network hyperexcitability which is proposed as present in DLB pathophysiology (Morris et al., 2015), and has been observed in Thy1-aSyn as well as in mice overexpressing rare α-syn mutations (Morris et al., 2015; Singh et al., 2019).