It can also be promoted by DC stimulators (such as Flt3, OK-432, GM-CSF) and by modifying the influence of tumor-associated antigens by TLR agonists (such as CpG, CpG-ODN, imiquimod, BCG, BCG-CWS) and agonists of costimulatory receptors (anti-CD40). The gene discussed is FLT3; the disease is neoplasm.