Myricetin inhibits NLRP3 inflammasome assembly by promoting non-reactive oxygen species (ROS)-dependent NLRP3 ubiquitination and reducing ROS-dependent ASC ubiquitination; this blocks the interaction between ASC and NLRP3, inhibits ASC aggregation, and alleviates the sepsis-induced inflammatory response (73). Here, NLRP3 is linked to Sepsis.