SOCS3 and Salmonella Infections: We also found that genes in Group C were disproportionately impacted by loss of hnRNP M, with nearly half of all genes in the category (40/98) upregulated in hnRNP M knockdown macrophages at 4h post-Salmonella infection, including the chemokine receptor Ccrl2, the regulator of NFκB Nfkbiz, and the repressor of JAK/STAT signaling Socs3.