Consistent with our observation that many DEGs identified in uninfected macrophages “lose reliance” on SR/hnRNPs upon Salmonella-infection, the only pathway that was significantly enriched for DEGs in both conditions was the translation/mTOR related pathway “Regulation of eIF4 p70S6K,” which remained significantly enriched for DEGs in SRSF6, SRSF9, and hnRNP F in Salmonella-infected macrophages (Figure 2B). This evidence concerns the gene SRSF9 and infection.