BCAP31 and Hyperglycemia: As shown in Figures 3A–C, maintenance of mitochondrial morphology protein 1 (Mmm1), mitochondrial distribution and morphology protein 34 (Mdm34), and B cell receptor-associated protein 31 (BAP31) were significantly upregulated in hyperglycemia-treated cardiomyocytes, suggesting that hyperglycemic stress increases mitochondria–ER contact.