The presence of the c.672∗26C > T, c.672∗47G > A, and c.672∗51G > T mutations within the 3′−UTR of SIGMAR1 affect transcript stability resulting in increased Sigmar1 transcript in human neuroblastoma SK−N−MC and HEK-293 cells (Luty et al., 2010). Here, SIGMAR1 is linked to neuroblastoma.