NR2F6 and hepatoblastoma: From molecular function annotation, these genes were shown to participate in the activity of serine-type endopeptidase, serine-type peptidase, serine hydrolase, et al. Further, as suggested by both KEGG and Reactome pathways (Table 2; Figure 7(c,d)), the upregulated NR2F6 and its targets notably exert a crucial influence on metabolism-related pathways for residual hepatoblastoma, especially cholesterol metabolism, retinoid metabolism and transport, and lipid-soluble vitamins metabolism.