Also, retinal organoids serve to model X-linked juvenile retinoschisis [105], and late-onset retinitis pigmentosa [106] and recapitulate the pathogenesis of retinitis pigmentosa, allowing for CRISPR-Cas9-mediated correction of RPGR mutation to reverse ciliopathy and photoreceptor loss [107]. Here, RPGR is linked to retinitis pigmentosa.