MEN1 and neoplasm: These preliminary and promising results, obtained in basic research, show the ability of the anti-miR-24-1 antagomir to target and inhibit the human endogenous miR-24-1 and restore the normal expression of wild type menin, and prompt the possibility to develop and design RNA antagomir-PEI-PMNP complexes against hsa-miR-24, for systemic administration to MEN1 patients and prevention of tumor development.