In addition to miR-24, other miRNAs showed to be possibly involved in the MEN1 tumorigenesis or to be deregulated in the sporadic tumor counterparts of the neuroendocrine tissues commonly affected in MEN1 syndrome [25], and they should be further investigated by functional studies, as was done for miR-24, to assess their importance in MEN1-related tumor development and progression. Here, MEN1 is linked to multiple endocrine neoplasia type 1.