LMNA and familial partial lipodystrophy: The latter was performed in a study by Boschman and co-workers [111], who observed markedly increased but incomplete skeletal muscle fatty acid oxidation, in combination with reduced glucose oxidation in patients with LMNA-related familial partial lipodystrophy (FPLD) and LGMD1B, in vivo and in vitro, in cultured myotubes derived from patients.