A study by Rzymski and colleagues reported that Senexin B potently suppressed phosphorylation of STAT5 Ser726 and STAT1 Ser727 in AML cell lines KG-1, HL-60, MOLM-16, MV-4-11, OciAML-2, and MOLM-6 [140]. The gene discussed is STAT1; the disease is acute myeloid leukemia.