Because Oxtr and Magel2 are co-expressed in aCA2/CA3d hippocampus in infancy (P7), and because in Oxtr KO mice [36], as in several models [38] of autism, the depolarizing to hyperpolarizing developmental GABA shift is delayed, we investigated the GABA-shift timing in Magel2-KO pups. The gene discussed is OXTR; the disease is autism.