The results showed that the eutopic and ectopic endometrium of adenomyosis patients treated with mifepristone showed significantly lower expression of B7-H2, B7-H3, B7-H4 and PD-L2 protein compared with that of adenomyosis patients without mifepristone treatment, both in the proliferative phase and in the secretory phase (Figs. 1, 2, 3 and 4g-j all P < 0.01). The gene discussed is PDCD1LG2; the disease is adenomyosis.