Recurrent gene fusions are seen in 93% of CRLF2‐overexpressed Ph‐like ALL patients,3 with many fusions inducing small deletions at the breakpoint that are detectable by high‐resolution chromosome genomic array testing (CGAT), also known as single nucleotide polymorphism (SNP)‐array comparative genomic hybridization. This evidence concerns the gene CRLF2 and acute lymphoblastic leukemia.