In addition, the percentage of NF-κB-p65 nuclear translocation in OFTu cells infected with OV-SY17Δ120 for 4 h was dramatically decreased compared with that of the OV-SY17- and OV-SY-RV120-infection groups (Fig. 6D), which ruled out the possibility that the mild phenotype caused by the deletion of ORF120 is a simply delayed activation of NF-κB signaling. The gene discussed is NFKB1; the disease is infection.