As, unsurprisingly, we did not detect an inducible mesenchymal-to-epithelial shift in A204 cells due to the non-epithelial origin of rhabdoid tumor, the strong induction of EGFR by HDAC inhibition rather represents a resistance mechanism, leading to a ‘kinase switch’ to sustain mesenchymal dedifferentiation. This evidence concerns the gene EGFR and rhabdoid tumor.