In vitro simulation of cell–cell interaction of tumor T lymphoblasts with APCs was accompanied by enhanced survival of CD4+, CD4+CD71+, and CD4+CD95+ subsets of CD45+CD3+ Jurkat T cells (Table S2) as well as a modified spectrum of the produced biomolecules with a statistically significant increase in VEGF and MIP-1β (CCL4 chemokine) concentrations (Table S3) that promotes cancer cell proliferation and metastasis [70,71]. This evidence concerns the gene CCL4 and neoplasm.