CTH and acute myeloid leukemia: While no consistent similarities between AML and CML were identified, dysregulation of genes involved in sustaining glutamate (i.e., PRODH), aspartate (i.e., AST1), alanine (i.e., GPT), asparagine (i.e., ASNS), and cysteine (i.e., CTH) levels suggest potential vulnerabilities involving these amino acids.