METTL3 and gastric cancer: For example, in gastric cancer, it has been reported that the methyltransferase METTL3 was essential for epithelial-mesenchymal transition (EMT) and metastasis, by enhancing ZMYM1 stability by increasing its m6A level to repress the transcription of E-cadherin (16); another methyltransferase KIAA1429 could enhance c‐Jun expression by stabilizing its mRNA in an m6A-dependent manner to promote cell proliferation (17).