Similarly, gene expression levels of ZFP36L2 in peripheral blood mononuclear cells are significantly lower in SLE patients than those in healthy controls (50). Therefore, the variants of ZFP36, ZFP36L1, and ZFP36L2 or dysregulation of those expressions may be involved in developing various inflammatory diseases in humans. Here, ZFP36L2 is linked to systemic lupus erythematosus.