Using publicly available data of GC-growth-arrested cervical cancer cell line model (HeLa) that upregulate CDKN1C upon GC treatment (Supplementary Fig. 7a–c), we observed binding of GR and multiple SWI/SNF members to the CERES enhancer (Supplementary Fig. 7d), suggesting that this mechanism is active in other cancer types. Here, NR3C1 is linked to cervical carcinoma.