The type 1 diabetes group had attenuated mobilization of HPCs and EPCs, ranging from 39 to 55% lower across the phenotypes when compare to the non-diabetes group, with CD34+ HPCs (331 ± 437 Δ cells/mL vs. 734 ± 876, Δ cells/mL p = 0.048) and CD34+VEGFR2+ EPCs (171 ± 342 Δ cells/mL vs. 303 ± 267 Δ cells/mL, p = 0.006) significantly lower. The gene discussed is KDR; the disease is diabetes mellitus.