Finally, the present findings as observed in rodents may extend to the human setting, considering that the KLR cocktail also prevents lipid-induced insulin resistance in hiPSC-CMs by v-ATPase1 reassembly – offering the potential of these AAs to be used as nutraceuticals to limit lipid uptake in diabetics with cardiomyopathy. The gene discussed is KLRK1; the disease is diabetes mellitus.