CRIP1 and gastric cancer: The results of MTT (3-(4, 5-dimethylthiazolyl-2)-2, 5-diphenyltetrazolium bromide) (Fig. 2E and Supplementary Fig. S2F), cell colony formation (Fig. 2F and Supplementary Fig. S2G), and flow cytometry assays (Fig. 2G, H and Supplementary Fig. S2H–I) all showed that EPI and CDDP both exhibited stronger cytotoxicity in GC cells with CRIP1 silencing, suggesting that CRIP1 is important for maintaining genome stability and is required for cell survival following DNA damage.