MerTK activation is involved in the suppression of inflammation, synthesis of inflammatory mediators, and promotion of apoptotic cell clearance following acute tissue damage during tissue repair.41–44 Triantafyllou et al.41 found that MerTK-expressing hepatic macrophages promote the resolution of inflammation in acetaminophen (APAP)-induced ALI, and that APAP-treated MerTK−/− mice exhibit persistent liver injury and inflammation. The gene discussed is MERTK; the disease is acute respiratory distress syndrome.