TNFRSF1A and tuberculosis: Within the TB context, reports suggest that TNFRs control exacerbated inflammation, whereas TNFR1 is necessary to recruit immune cells at the mycobacterial infection site, and tmTNFR2 expression is necessary to induce the regulatory function in myeloid-derived suppressor cells (Chavez-Galan et al. 2016, 2017, 2019; Uysal et al. 2018).